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	<title>MY HEALTH RIGHTS &#187; Sporotrichosis</title>
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		<title>THE DEADLY SIDE OF FUNGAL EXPOSURE</title>
		<link>http://www.myhealthrights.com/2008/06/fungalexposure/</link>
		<comments>http://www.myhealthrights.com/2008/06/fungalexposure/#comments</comments>
		<pubDate>Fri, 27 Jun 2008 20:13:37 +0000</pubDate>
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				<category><![CDATA[Morgue]]></category>
		<category><![CDATA[and skin diseases. toxic fungi]]></category>
		<category><![CDATA[Aspergillosis]]></category>
		<category><![CDATA[Blastomycosis]]></category>
		<category><![CDATA[Candidiasis]]></category>
		<category><![CDATA[Chromoblastomycosis]]></category>
		<category><![CDATA[Coccidioidomycosis]]></category>
		<category><![CDATA[Cryptococcosis]]></category>
		<category><![CDATA[eye infections]]></category>
		<category><![CDATA[FUNGAL EXPOSURE]]></category>
		<category><![CDATA[hair]]></category>
		<category><![CDATA[Histoplasmosis]]></category>
		<category><![CDATA[immune system]]></category>
		<category><![CDATA[infections]]></category>
		<category><![CDATA[Lobomycosis]]></category>
		<category><![CDATA[mold contamination]]></category>
		<category><![CDATA[mold exposure]]></category>
		<category><![CDATA[Mycetoma]]></category>
		<category><![CDATA[nail]]></category>
		<category><![CDATA[Otomycosis]]></category>
		<category><![CDATA[Paracoccidioidomycosis]]></category>
		<category><![CDATA[Penicilliosis marneffeii]]></category>
		<category><![CDATA[Phaeohypomycosis]]></category>
		<category><![CDATA[Rhinosporidioisis]]></category>
		<category><![CDATA[Sporotrichosis]]></category>
		<category><![CDATA[Zygomycosis]]></category>

		<guid isPermaLink="false">http://www.myhealthrights.com/?p=24</guid>
		<description><![CDATA[Little black spots begin to speckle the wall, as a musty smell fills the room winding its way into your unsuspecting nostrils.  You walk across your living room carpet with bare feet and sense an unwelcome sensation of moistness seeping between your toes.  Suddenly realizing that water is penetrating the security of your home like [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: x-small; font-family: Arial;">Little black spots begin to speckle the wall, as a musty smell fills the room winding its way into your unsuspecting nostrils.  You walk across your living room carpet with bare feet and sense an unwelcome sensation of moistness seeping between your toes.  Suddenly realizing that water is penetrating the security of your home like a burglar in the night, your first impulse is a sense of low-level panic!  Where is the moisture coming from  Then, it hits you like a freight train.   Those specks on the wall are mold! </span></p>
<p class="MsoNormal"><span style="font-size: x-small; font-family: Arial;">Frantically, you begin searching for the source of leakage into your home.  Lifting up the carpet, you notice water is all over the floor-base and sections of the wood molding are dry while one long patch under the speckled portion of the wall is completely saturated.  The problem could be that the actual leakage is coming from within the wall and not from an easily detectable area such as a patio doorway.  You wonder how long this has been happening and what can be done about itfast!  You and your wife are elderly and maneuverability is difficult, so tackling this problem could be beyond your physical capabilities.  What can you do </span></p>
<p class="MsoNormal"><span style="font-size: x-small; font-family: Arial;">You and your wife have been having sinus problems over a six-month period with constant attacks of flu-like symptoms.  Is there a connection between your discovery of mold contamination and the health problems you are experiencing  Is mold exposure hype or really dangerous <span id="more-24"></span></span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">According to research offered by the Medical Mycology Research Center, Department of Pathology, University of Texas Medical Branch, there are several known diseases and/or infections caused by human exposure to toxic fungi.  Some of these are: </span></p>
<p class="MsoBodyText" style="text-indent: 0.5in;"><span style="font-size: x-small; font-family: Arial;"> <strong>Aspergillosis, Blastomycosis, Candidiasis, Chromoblastomycosis, Coccidioidomycosis, Cryptococcosis, Histoplasmosis, Lobomycosis, Mycetoma, Otomycosis, Paracoccidioidomycosis, Penicilliosis marneffeii, Phaeohypomycosis, Rhinosporidioisis, Sporotrichosis, Zygomycosis.</strong>  Also, <strong>eye infections, hair, nail, and skin diseases. </strong></span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">This is not isolated research, but what does it all mean  Compelling research offered from reputable laboratories throughout the world strongly supports the fact that fungi exposures can be cause conditions dangerous to human health, which can lead to death. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">Detecting water intrusion that results in fungal growth, especially in a dwelling occupied by the elderly should never be handled personally.  This is a job for professional environmental hygienists who test for contamination levels of various fungi and remediation experts who repair and restore contaminated sites.  After the age of 30, the human immune system gradually becomes weaker and so a person who is elderly may have fixed the problem, but also could very likely cause health problems that can lead to very costly medical expenses.  Is it worth it </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;"> The first remedy in finding a solution is to check your Homeowners Insurance Policy for coverage relating to plumbing and building construction problems and then call your insurance provider for more information or a home inspection.  If you rent, or your home is owned by someone else, inform your landlord of the mold contamination and insist it be explored and remediated it to your satisfaction promptly.  Normally, you do not have to become angry to get things done, but be firm.  Some fungi are able to grow and multiply within 24-48 hours under compatible conditions.  Many types of fungi occupy your home already and are found in carpeting, heating and air conditioning units, air ducts, and in other areas where they can go undetected.  Several types are also in your body living normally and even helping your immune system.  They are referred to as opportunistic fungi, and from time to time they can disturb your body, but your immune system keeps them in-check enough to work in harmony with natural metabolism. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">Generally, when indoor fungi (or their reproducing particles called spore) are approximately two times or more greater in comparison to outdoor levels, and indoor humidity is often greater than 50 percent consistent with warmer indoor temperatures anywhere from any low-level temperature up to about 90 degrees, fungi contamination is possible. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">The consensus of many environmental authorities indicates that humidity levels lower than 30-45 percent combined with normal room temperature settings at approximately 70 degrees, where water infiltration is not evident, is a somewhat safer environmental condition. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">The idea of this article of course is not to place any fear into the hearts of anyone, but to inform people in this particular age group of the health problems involved with a fungi-contaminated environment and that if detected and corrected effectively, this whole problem can be prevented.  It does not have to be a health problem, but if it is, very few medical doctors we have consulted know of, or even want to be involved in treating it.  The associated costs of medicine and treatment are astronomical monthly and can range anywhere from two to five thousand dollars depending on the severity and complications involved!           </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">Due to advanced medical research capabilities, fungal infections have emerged as a major cause of morbidity and mortality, particularly in persons with cancer, AIDS, or disorders of the human immune system. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">There are certain immune disorders to be concerned with characterized by fungal ailments, these are: <strong>Autoimmune disease, an Immune Compromised or Suppressed system, and/or an Immune Deficient or Depressed system.<span style="font-size: 11pt; font-family: Century Gothic;">  </span></strong>   </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">  </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;"><strong>Autoimmune disease</strong> is a medical condition characterized by the subversion or alteration of the function of the immune system of the body, resulting in the production of antibodies against the bodys own cells treating them as if they were external intruders (pathogens) attempting to invade the body.  Some recognized autoimmune diseases include: </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">  </span></p>
<p class="MsoBodyText" style="margin-left: 0.5in;"><strong><span style="font-size: x-small; font-family: Arial;">Addisons disease (affecting the adrenal gland) Autoimmune hemolytic anemia (affecting red blood cell membrane proteins), Chrons disease (gut), Graves disease (a toxic nodule goiter associated with hyperthyroidism), Hashimotos disease (goiter associated with hypothyroidism), Idiopathic thrombocytopenic purpura (platelets), Insulin dependent diabetes mellitus I (pancreatic beta cells), Multiple sclerosis (affecting brain and spinal cord neural systems), Myasthenia gravis (affecting nerve and muscle synapses), Pemphigus vulgaris (affecting skin), Pernicious anemia (gastric parietal cells), Poststreptococcal glomerulonephritis (kidney function), Psoriasis (skin rashes), Rheumatoid arthritis (connective tissue), Scleroderma (affecting heart, lungs, gut, and kidneys), Sjns syndrome (affecting liver, kidneys, brain, thyroid, and salivary gland), Spontaneous infertility (Sperm), and Systematic lupus erythematosus (affecting DNA, platelets and other tissues). </span></strong></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">  </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">An <strong>immune compromised or suppressed system</strong> can occur as the result of an injury or medical surgical operation that has required the natural immune response to overproduce and then heal over time. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">An <strong>Immune deficient or depressed system</strong> can occur as the result of an ailment acutely (suddenly) or chronically (lingering or recurring) affecting a person that could be implicative of major medical problems that the natural immune system is constantly having to fighting off.  (Conditions involved might include flu symptoms, colds, sinus problems, allergies, etc.) </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">Generally, any condition or illness relating to the immune system over an extended period suggestive of one or more weeks should involve professional medical attention.  This is just good common sense. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">Much has been said and written about the Black Mold, or a fungus called Stachybotrys chartarum (atra), and how dangerous it can be in a contaminated environment.  According to research conducted at the University of Minnesota, as follows: </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">  </span></p>
<p class="MsoBodyText" style="margin-left: 0.5in;"><strong><span style="font-size: x-small; font-family: Arial;">The [multiple] potent mycotoxins [trichothecenes and atranones] produced by the fungus [Stachybotrys chartarum] will suppress the immune system affecting the lymphoid tissue and bone marrow.  Animals injected with the mycotoxin from this fungus exhibited the following symptoms: necrosis (localized tissue death that occurs in groups of cells response to tissue disease or injury) and hemorrhage within the brain, thymus, spleen, intestine, lung, heart, lymphnode, liver, and kidney. </span></strong></p>
<p class="MsoBodyText" style="margin-left: 0.5in;"><strong><span style="font-size: x-small; font-family: Arial;">  </span></strong></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">Affects by the absorption of mycotoxins (or fungal secondary poisonous chemicals) on the lungs, is referred medically as pneumomycosis.  Most of the attention given to the Stachytbotrys mold rests on investigative studies involving infant deaths, cancer patients, and severely ill HIV/AIDS patients, and little that we could find has been directed toward the elderly. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">Research data compiled by the Albert Einstein College of Medicine attained at a medical symposium held on January 29, 2000 at the MD Anderson Cancer Center in Houston, Texas, and information now certified for continuing education of physicians by the Accreditation Council for Continuing Medical Education (ACCME), indicates the following alert to medical professionals in its training curriculum: </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">  </span></p>
<p class="MsoBodyText" style="margin-left: 0.5in;"><strong><span style="font-size: x-small; font-family: Arial;">Mortality is higher when the diagnosis of fungal infection is not made early.  In one study, patients with lymphoma were housed in a protective environment &#8212; single-patient rooms under positive pressure with high-efficiency particulate air filtration&#8211; while undergoing bone marrow or peripheral stem cell transplantation. </span></strong></p>
<p class="MsoBodyText" style="margin-left: 0.5in;"><strong><span style="font-size: x-small; font-family: Arial;">  </span></strong></p>
<p class="MsoBodyText" style="margin-left: 0.5in;"><span style="font-size: x-small; font-family: Arial;"><strong>Despite these protective measures, 5.2% of patients developed nosocomial [hospitalization-related] aspergillosis.  Although mortality rates among the patients with localized or pulmonary disease were substantial (42%), they were better than those of the patients who began treatment after the disease was already disseminated [spread to specific organs and/or tissues of the body).  In fact, none of the patients with documented disseminated disease survived. </strong></span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">  </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">Immunosuppressed persons, persons with known autoimmune disease(s), and/or immune compromised persons who develop new or progressive pulmonary infiltrates while receiving broad-spectrum antibiotics, present major problems for differential diagnosis.  These people are prime candidates for combined opportunistic fungal, bacterial, and viral infections and diseases with multiple medical complications.  But, some evidence has surfaced recently that non-immune impaired individuals could also be at risk. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">It is imperative that physicians in all disciplines of medicine consider current research findings <strong>(preferably starting with the 1999 Merck Manual, 17<sup>th</sup> Edition, Chapter 158, Pages 1209-1226, Systemic Fungal Diseases/Systemic Mycoses)</strong>, in conjunction with any case where high-level fungal exposures are suspected.  In the past, it could be that some or even most medical doctors tend to discount this and as a consequence often the serious illnesses are not immediately recognized or properly diagnosed.  The only answer to this medical field phenomena would be speculative at best since some of the worlds most prominent experts have written on this subject, so hopefully medical doctors will begin to enlighten themselves with future symposiums on the subject.  Also, the Centers for Disease Control and other government agencies might want to be highly supportive in their budgets for more dedicated research in this vital human interest medical campaign. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">One thing is very accurate.  Fungal microorganisms in being Eukaryotic, or single-celled organisms containing a distinct membrane bound by a nucleus observed in all higher organisms are considered neither animal nor plant cells.  This is why they have their own Kingdom as a species occupying the earth. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">Unlike plants, fungi cannot make their own food, and must absorb it from dead organic matter or other living organisms.  When fungi nutrition is derived from dead mater it is called substrate, and from a living entity it is called the host.  Hence, a human being is a human host to the fungi eating what it eats. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">When healthy, the fungi as stated earlier live in harmony with their human host, but if nutrition becomes scarce or this delicate balance disrupted by a weaker pathogen, e.g., Stachybotrys that is not normally found in the human host the trouble starts.   Neither bacteria nor viruses are similar to fungi in composition, but fungi lean characteristically more toward functioning as animal cells. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">If an outside organism such as a flea, mosquito, or tick were to inflict their toxins into a human this would certainly be easier to detect in preventing illness.  Likewise, if a person should run across an orange, banana, or other vegetation covered with fungi a healthy person would not want to eat it  </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">Some forms of mushrooms and plants are deadly when eaten.  This is how they protect their species ecologically in survival.  These mushrooms are fungi very similar to their microscopic counterparts.  The basic differences lie with size, capacity, and metabolism.  The question that arises is that if everything in the natural sciences conclusively demonstrates this, why is it so difficult to understand it medically  Thousands of elderly people will be medically treated for sinusitis this year with antibiotic drugs that could be potentially harmful to their immune systems if fungal exposures are at the base of their illness.  Most antibiotics do not work with fungal disorders. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">Hopefully soon, national health legislation in congress will begin to address both the medical training that is lacking, proper treatment, and research that is greatly needed to make medical professionals more aware and accountable for any misdiagnosis. </span></p>
<p class="MsoNormal"><span style="font-size: x-small; font-family: Arial;">The following precautionary alert was offered to physicians attending the symposium: </span></p>
<p class="MsoNormal" style="margin-left: 0.5in;"><strong><span style="font-size: x-small; font-family: Arial;">There are no rapid, accurate diagnostic tests that can confirm with certainty the presence of invasive fungal disease.  Unless the clinician considers fungal disease early, disease can progress rapidly while the patient is treated aggressively with broad-spectrum antibiotics.  Not only are fungal infections difficult to distinguish from bacterial [because bacteria are environmentally-adaptive to fungi] or other infections, but the clinical manifestations of many fungal infections are shared among a variety of fungal pathogens as well. </span></strong></p>
<p class="MsoNormal" style="margin-left: 0.5in;"><strong><span style="font-size: x-small; font-family: Arial;">  </span></strong></p>
<p class="MsoNormal" style="margin-left: 0.5in;"><span style="font-size: x-small; font-family: Arial;"><strong>Standard microbiology is often adequate to provide diagnosis.  Short of tissue biopsies, fungal cultures are not always positive in the presence of invasive disease.  Moreover, positive cultures do not definitively signify invasive disease; they may be represent colonization.  Nonetheless, in patients who are immunosuppressed, a positive culture and invasive disease are highly correlated.  A high-risk patient with a positive culture should be considered to have invasive disease until proven otherwise.</strong><span style="font-size: 12pt; font-family: Century Gothic;">   </span></span></p>
<p class="MsoNormal"><span style="font-size: x-small; font-family: Arial;">  </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">According to several laboratory studies involving the study of antibiotic medicines, persons suffering from fungal (mycotic) infections are often misdiagnosed and the continued use of antibiotics is shown to suppress the natural immune system in fighting disease. </span></p>
<p class="MsoNormal"><span style="font-size: x-small; font-family: Arial;"> Dr. Luther E. Lindner, a Pathologist on the faculty of Texas A&amp;M University, College of Medicine and involved with private research with Pathobiotek Diagnostics, Inc., located in Houston, Texas indicates the following according to his research on antibiotics:  </span></p>
<p class="MsoNormal"><span style="font-size: x-small; font-family: Arial;"> We have found that under certain circumstances antibiotics can actually stimulate bacterial [fungal] growth and make the patient worse</span></p>
<p class="MsoNormal"><span style="font-size: x-small; font-family: Arial;">It is common and well-established medical knowledge that over use or inappropriate use of antibiotic can increase the number of resistance factors in our environment.  A full course of antibiotics prescribed is chosen to maximize the destruction of the targeted pathogen and minimize the generation of resistance factors.  Today, in science, there is an ongoing race between the wealth of genetic material available to control bacteria and/or fungi and in the antibiotics being discovered and adapted by people. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">The black mold Stachybotrys chartarum, for which Center for Disease Control has issued a statement requiring further investigation, is documented scientifically as a known cause of immunosuppression in otherwise healthy people!  This is an alarming observation because the health risks of contamination in the elderly of great concern now! </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">Recent medical research from the Mayo Clinic in Rochester, Minnesota implicates fungus as the cause of chronic sinusitis, in as many as 96 percent of the cases.  In researching 210 patients known to have chronic sinusitis, they identified an average of 2.7 kinds of fungi exposure.  They also identified over 40 variants of fungi species during their controlled research project. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">Scientific research using sophisticated and highly technical advances into human pathology is very specific about the destructive molecular health consequences that antigens cause in known autoimmune diseases mentioned in current medical studies. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">Scientific American magazine devoted its September 1993 issue to this research and listed Graves Disease (and Hashimotos disease as well) as an autoimmune disease.  <strong>Reference: Scientific American magazine, September 1993, Author; Lawrence Steinman, Professor of Neurological Science and Pediatrics, Stanford University; Title, Autoimmune Disease, Research experiments, Daniel P. Gold, San Diego Regional Cancer Center, and Halina Offner and Arthur A. Vandenbark, Oregon Health Sciences University School of Medicine, Pp. 109. </strong></span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">  </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">In an animal model of multiple sclerosis, an autoimmune disease called Experimental Allergic Encephalomyeltis (EAE), researchers proved the value of the model, which is induced by autoimmunizing animals against myelin basic protein and which produces the clinical symptoms of multiple sclerosis: paralysis and demyelination.  The model can thus test therapies for the human illness.  In this experiment, the following was postulated: </span></p>
<h5><span style="font-size: x-small; font-family: Arial;">  </span></h5>
<h5 style="margin-left: 0.5in; text-indent: -0.5in;"><span style="font-size: x-small; font-family: Arial;">            <strong>In autoimmune disease, what turns the <em>T</em> cells against the self  Infection [recall that bacteria and mold growth are normally found together which are causation of infections] often precedes the onset of autoimmune disease, and so scientists have closely scrutinized the tactics that pathogens commonly employ to elude <em>T</em> cells.  The answer appears to lie in molecular mimicry, and evolutionary adaptation whereby viruses and bacteria [and, therefore toxiciogenic fungi also, because it is the oldest and chemically strongest in adaptive microorganism survival, feeding viruses and bacteria] attempt to fool the body into granting them free access.  Such mimicry works by showing the immune system stretches of amino acids that look like self.  For example, adenovirus type 2 has amino acid sequences like those in the crucial fragment of myelin basic protein.  In responding routinely to this virus, the immune system may become primed to attack the corresponding self-component myelin.  An autoimmune response can begin even if the molecular mimicry is not quite exact.</strong> </span></h5>
<h5><span style="font-size: x-small; font-family: Arial;">  </span></h5>
<h5><span style="font-size: x-small; font-family: Arial;">According to the article, <strong>This research suggests that molecular mimicry between viruses or bacteria [to include toxiciogenic fungi] and self may be critical in initiating autoimmune responses.</strong>  </span></h5>
<h5><span style="font-size: x-small; font-family: Arial;">This research also tends to explain why exposures to Aspergillus and Penicillium are usually undetectable in blood serum tests, and why DNA tests of these deadly fungi in the biological system are required in order to conclusively detect their mimicking presence within the immune system. </span></h5>
<h5><span style="font-size: x-small; font-family: Arial;">Stachybotryotoxicosis documentation researched by Harvard School of Public Health, Michael L. Muilenberg, Research Associate/Instructor, indicates: </span></h5>
<h5 style="margin-left: 0.5in;"><strong><span style="font-size: x-small; font-family: Arial;">Some strains of Stachybotrys [chartarum] produce potent toxins with a variety of health effects.  Particularly nototious, are cases in the Ukraine about 60 years ago [actually the late 1920s] where livestock, especially horses, became very sick, developing ulcers around the mouth, inflammation of the respiratory track, fever, and blood clotting failure.  Many died within days to weeks after onset of symptoms.</span></strong><span style="font-size: x-small; font-family: Arial;"> <strong>A number of similar cases (including a case involving sheep in Hungary) have been reported since that time.</strong> </span></h5>
<h5><span style="font-size: x-small; font-family: Arial;">  </span></h5>
<h5><span style="font-size: x-small; font-family: Arial;">In addition, Michael Muilenberg continues: </span></h5>
<h5><strong><span style="font-size: x-small; font-family: Arial;">The toxins which can be produced by this fungus are classified as [macrocylic] trichothecenes [highly toxic compounds] and include; satratoxin[s] f, g, h, [trichoverrols/trichoverrins], verrucarin J, and roridin E.  Symptoms of exposure can include skin and respiratory tract inflammation, fever, malaise, immunosuppression, among others.</span></strong><span style="font-size: x-small; font-family: Arial;"> </span></h5>
<p class="MsoNormal"><span style="font-size: x-small; font-family: Arial;">The Clinical Pharmacology and Nursing Management manual Second Edition (1986), Systemic Fungal Infections Pg. 307, indicates the following: </span></p>
<p class="MsoNormal" style="margin-left: 0.5in;"><strong><span style="font-size: x-small; font-family: Arial;">Although systemic fungal infections are serious, life-threatening conditions, most escaped notice until relatively recently due to misdiagnosis.  The lung diseases, blastomycosis, coccidioidomycosis, and histoplasmosis, mimic tuberculosis so closely that they could not be differentiated from that condition.  When effective tuberculosis chemotherapy was developed, some tuberculosis clients who failed to respond to the new drugs were found to be suffering from entirely different diseases the fungal infections.  Fungi can affect tissues other than the lungs, causing pleural, peritoneal, <span style="text-decoration: underline;">ocular</span>, <span style="text-decoration: underline;">urinary</span>, and <span style="text-decoration: underline;">meningeal </span>infections. </span></strong></p>
<p class="MsoNormal"><span style="font-size: x-small; font-family: Arial;">  </span></p>
<p class="MsoNormal"><span style="font-size: x-small; font-family: Arial;">The medical manual Human Pathology; An Introduction to the Study of Disease Author: David T. Rowlands, Jr., M.D. (1986), Neoplasms Pg. 383, Paragraph 1, indicates the following: </span></p>
<p class="MsoNormal" style="margin-left: 0.5in;"><strong><span style="font-size: x-small; font-family: Arial;">Aflatoxins, metabolic products of saprophytic fungi (e.g., Aspergillus flavus) known to be hepatic carcinogens, are especially common in parts of Africa and Asia, where hepatomas are most often seen.  It is also possible that cirrhosis itself may encourage hepatoma formation.  The hepatitis virus, aflatoxins, and cirrhosis have in common prolonged inflammation of the liver.  None of these are, by themselves clearly causative.  Hormonal stimulation is important in some cases, since hepatomas have been recognized in patients on sustained androgen therapy.</span></strong><span style="font-size: x-small; font-family: Arial;"> </span></p>
<p class="MsoNormal"><span style="font-size: x-small; font-family: Arial;">  </span></p>
<h5><span style="font-size: x-small; font-family: Arial;">In summing up all of this, when symptoms and medical indicators provide evidence of fungal contamination at high levels of exposure in the human body, it is very evident that health is greatly disturbed, and time is short for finding answers and effective treatments.</span></h5>
<p class="MsoNormal"><span style="font-size: x-small; font-family: Arial;">Doctors who are not well informed or continue to disbelieve that fungi exposure can threaten human life often pick, probe, harass, and in some cases even delay any promise of swift medical treatment.  As a patient, or even a concerned person wanting answers your best hope at present is to obtain medical advisement and assistance from those few medical professionals who exist whom are most appropriately equipped with the specific knowledge and experience to research and properly diagnose this medical condition.  This help is available mainly through Environmental and Occupational physicians preferably specializing in allergy and immunology at this juncture. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">If you suspect fungi contamination do not delay in contacting professional help in eliminating the potential for illness.  Seek medical attention early in evaluating whether or not fungi contamination is adversely affecting your health, but above all do not hesitate.  If your doctor indicates that he or she does not really understand or know much about fungal illnesses, ask to consult with a specialist who does.  The idea is not to contradict or offend your doctor, but to team up in ruling out the possibility that fungal exposure is adversely affecting your health. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">We have found that a blood-serum test cultured with agar agents known as the Multiple Antigen Simultaneous Test (MAST) CLA, Environmental IgG, Panel #15, conducted by Hitachi Chemical Diagnostics, Incorporated of Mountain View, California, and a genetic Polymerase Chain Reaction (PCR) testing process conducted by ImmunoLabs, Inc., in the Los Angeles, are both very helpful in detecting fungi levels in the human body. </span></p>
<p class="MsoBodyText"><span style="font-size: x-small; font-family: Arial;">There are also certain blood-serum tests available through blood laboratories for the Stachybotrys species.  These can be expensive.  Consult with, and work through your medical doctor if you are going to have them ordered. </span></p>
<p><span style="font-size: x-small; font-family: Arial;">According to several authorities involved with mycotic (fungal) disease investigation, serum tests rarely provide any clear evidence of contamination from the two most notorious destructive and deadly opportunistic fungi strains, those being Aspergillus (fumigatus, flavus, niger, etc.), and Candida albicans.  Normally either a biopsy or more recently DNA tests are most effective for optimal diagnosis.  Remember, you are the only one who is able to understand and relate to your body.</span></p>
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		<title>MOLD MYTHS AND LEGENDS- Microbial Mold Pathogens</title>
		<link>http://www.myhealthrights.com/2008/05/mold-myths-and-legends-microbial-mold-pathogens/</link>
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		<pubDate>Fri, 30 May 2008 18:35:23 +0000</pubDate>
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		<description><![CDATA[The myth that mold clustering and mycotoxins entering the human body through inhalation �are not� hazardous to health is absolutely absurd! Let�s evaluate this statement first, with common sense and without any conflicting science to clog our minds.
Given this, our initial question is most appropriate. What primary purpose do microbes, animals, and plants have on [...]]]></description>
			<content:encoded><![CDATA[<p>The myth that mold clustering and mycotoxins entering the human body through inhalation �are not� hazardous to health is absolutely absurd! Let�s evaluate this statement first, with common sense and without any conflicting science to clog our minds.<br />
Given this, our initial question is most appropriate. What primary purpose do microbes, animals, and plants have on earth? The answer is that they all contribute to activate a process called �dust-to-dust,� or if we want to add the scientific term to this it is it is called the, �carbon cycle.�<br />
What is the �carbon cycle� and what does it accomplish? The carbon cycle consists of steps by which carbon in the form of carbon dioxide, a gas produced by the oxidation (depletion of electron production) in carbon (one of the original building blocks of life) is released and recycled into the Earth�s atmosphere and is reused in the creation of life. Having become a living product of cell respiration (or, a process of the molecular exchange of oxygen and carbon dioxide within the body�s tissues) the carbon dioxide is extracted from and returned to the atmosphere by living, breathing and<span id="more-16"></span> excreting organisms. This cascade of events begins with photosynthetic productivity (or, an event by which green plants containing chlorophyll create chemicals such as carbohydrates out of water and carbon dioxide that float through the atmosphere using light as an energy source to release their byproduct oxygen, into the air. Animals and humans then again eat carbohydrates (sugars), which become fuel for every living cell in their biological systems. This process ends with the exhalation of carbon dioxide by these same beings.<br />
This same release of carbon dioxide also occurs during the decomposition (rotting) process that takes place after a plant or animal dies. While in a living state several other chemical processes interact with the ingestion of carbohydrates and the subsequent release of carbon dioxide into the atmosphere, and many of these same chemical processes inside the human body (i.e., enzymes, acids, and pH, etc.) actually protect against molds and other flora (i.e., bacteria, parasites, viruses) from starting to decompose the animal or human body before its time. This process works remarkably well as long as everything that is designed to function well continues to process according to its symbiotic state (or meaning, function and live in harmony).<br />
What on earth does all of this have to do with our opening statement? Well, molds according to genetic and molecular structure are quite similar to human cells in both function and physical characteristics. In fact, their composition offers more in similarity to their animal and human counterparts (even though they a far simpler form of life), than they do in comparison with any form of plant life. Molds also have one very complex and disturbing characteristic that sets them aside from bacteria, viruses, and many other microbes. They consist of a number of nuclear structures within their cells called �nuclei.�<br />
In fact some mold species have over 100 nuclei within their cell structure compared to the animal and human �single nucleus� cell. They also are able to withstand, and in fact frolic best in higher temperate and more humid climates than their animal �hosts.� Let�s face it, when our human body is measurably healthy with an immune system that is functioning well, molds and other flora throughout our biological system are very harmonious symbiotically, thrilled to live and flourish in a 98.6oF (370C) temperature, with an approximate 70 percent relative humidity rate throughout most of our human lifespan. Why would any mold species want to disturb something so comfortable? Well, the truth is, �once a pathogen, always a pathogen!� This is why they are called �opportunistic� pathogens. They do not want their comfortable animal or human environment to change, but when they �biologically sense� that metabolism is starting to lose its grip on agility, chemistry, or immunity through major invasion by other pathogens, especially other mold species that are not suppose to be traveling through or stopping by, they begin their natural �survival� process. Molds then become true pathogens in defending their territorial areas and often aggressively �cluster� into other areas once controlled by human chemistry that is now weakened or failing. Humans then also become their �prey!�<br />
Did we say �prey?� Why yes we did, and it was not a �slip of the tongue!� Almost every living thing and even things that are not living eventually become �prey� to molds- even bacteria, viruses, and other microbes. That�s right! In the ecological sub-world of microbes, molds are primarily designed to process the initial decomposition of a �deceased� entity, be it a plant or an animal species. That is what composts of rotting fruits and vegetation are made of, decaying rot initiated by none other than molds. And what do we extract from rotting fruits and vegetables? Alcohol, of course, is a byproduct of none other than some of these very molds! In fact, some of these molds are dangerous to animals and humans in high enough concentrations. This is why many constraints are placed on the agricultural production and commercial processing of beverages, tobacco, and food products we as humans consume.<br />
In some cases within even highly controlled hospital environments, some molds (again that are designed to destroy life to recreate new forms of life) can invade in much lower numbers, be inhaled by unsuspecting hospital staff, visitors, and patients alike, because molds cannot distinguish between these entities and have been known to cause �nosocomial� (or hospital related/caused) illnesses and sometimes death. In all fairness however, some species of bacterium can also cause similar diseases and death in a nosocomial or hospital setting. But if this is this true, could this be much more of a concern to healthy people who have much more air intake capacity than the often critically-ill patients we are speaking of in nosocomial situations?<br />
Let�s see what medical and laboratory students are told as they begin their studies in their class, Introduction to Diagnostic Microbiology, taught by Associate Professor and Director of Medial Laboratory Programs, Maria Dannessa Delost, M.S., M.T. (ASCP) of the Department of Allied Health, College of Health and Human Services, Youngstown State University. Professor Delost offers the following health and safety policies and procedures:<br />
�Conidia and spores may remain dormant in the air or environment or may be transported through the air to other locations. The spores of pathogenic molds can be inhaled and enter the respiratory tract. This is a common rout of infection, and because of this, it is imperative to practice good laboratory safety when working in mycology. All work, including the preparation of slides, plating and transferring cultures, and nay biochemical work, must be performed in a biological safety cabinet. Because airborne conidia and spores are readily released from a fungal culture, one should never smell a fungal culture. Screw-cap test tubes should be used in place of test tubes with a cotton, metal, or plastic lid. In addition, Petri plates must be sealed tightly with either an oxygen-impermeable tape or Parafilm. As always, gloves should be worn and any breaks or cuts in the skin covered to prevent the transmission of fungal infection. Now, why would an actively practicing and teaching scientist say this?<br />
Mycology, or the scientific study of the �Kingdom Fungi� is revealing many of the dark secrets that molds, members of the fungal kingdom, are giving up after thousands of years of literally living in complete silence. And, it is not very pleasant news! For instance, molds are far older than animals and humans and therefore have adapted genetically and metabolically to practically every illness, climatic change, and adverse situation known to life along the way. Animals and humans are in their early stages of existence compared to the Kingdom Fungi, and molds provide documented proof throughout history as to their ability to cause illness and death. Yet, because we are an �intellectual� human race I suppose, we continue to believe that we are much stronger than mold microbes that have �sensitized� to �read us like a book,� and can adjust rapidly to any environmental and metabolic change we as animals and humans encounter. Let�s see what medical doctors are telling other doctors about molds and what they are capable of when they become pathogenic to humans.<br />
According to research offered by the Medical Mycology Research Center, Department of Pathology, University of Texas Medical Branch, there are several known diseases and/or infections caused by human exposure to mycotoxins produced by some dangerous mold species. Some of these are:<br />
�Aspergillosis, Blastomycosis, Candidiasis, Chromoblastomycosis, Coccidioidomycosis, Cryptococcosis, Histoplasmosis, Lobomycosis, Mycetoma, Otomycosis, Paracoccidioidomycosis, Penicilliosis marneffeii, Phaeohypomycosis, Rhinosporidioisis, Sporotrichosis, Zygomycosis.� Also, �eye infections, hair, nail, and skin diseases.�<br />
This is not isolated research, but what does it all mean? Compelling research offered from reputable laboratories throughout the world strongly supports the fact that fungi exposures can be cause conditions dangerous to human health, which can lead to death.<br />
Due to advanced medical research capabilities, fungal infections have emerged as a major cause of morbidity and mortality, particularly in persons with cancer, AIDS, or disorders of the human immune system. There are certain immune disorders to be concerned with characterized by fungal ailments, these are: Autoimmune disease, an Immune Compromised or Suppressed system, and/or an Immune Deficient or Depressed system.<br />
Autoimmune disease is a medical condition characterized by the subversion or alteration of the function of the immune system of the body, resulting in the production of antibodies against the body�s own cells treating them as if they were external intruders (pathogens) attempting to invade the body. Some recognized autoimmune diseases include:<br />
Addison�s disease (affecting the adrenal gland) Autoimmune hemolytic anemia (affecting red blood cell membrane proteins), Chron�s disease (gut), Graves disease (a toxic nodule goiter associated with hyperthyroidism), Hashimoto�s disease (goiter associated with hypothyroidism), Idiopathic thrombocytopenic purpura (platelets), Insulin dependent diabetes mellitus I (pancreatic beta cells), Multiple sclerosis (affecting brain and spinal cord neural systems), Myasthenia gravis (affecting nerve and muscle synapses), Pemphigus vulgaris (affecting skin), Pernicious anemia (gastric parietal cells), Poststreptococcal glomerulonephritis (kidney function), Psoriasis (skin rashes), Rheumatoid arthritis (connective tissue), Scleroderma (affecting heart, lungs, gut, and kidneys), Sj�gren�s syndrome (affecting liver, kidneys, brain, thyroid, and salivary gland), Spontaneous infertility (Sperm), and Systematic lupus erythematosus (affecting DNA, platelets and other tissues).<br />
Much has been said and written about the �Black Mold,� or a fungus called �Stachybotrys chartarum (atra), and how dangerous it can be in a contaminated environment. According to research conducted at the University of Minnesota, as follows:<br />
�The [multiple] potent �mycotoxins� [trichothecenes and atranones] produced by the fungus [Stachybotrys chartarum] will suppress the immune system affecting the lymphoid tissue and bone marrow. Animals injected with the mycotoxin from this fungus exhibited the following symptoms: necrosis (localized tissue death that occurs in groups of cells response to tissue disease or injury) and hemorrhage within the brain, thymus, spleen, intestine, lung, heart, lymphnode, liver, and kidney.�<br />
Research data compiled by the Albert Einstein College of Medicine attained at a medical symposium held on January 29, 2000 at the MD Anderson Cancer Center in Houston, Texas, and information now certified for continuing education of physicians by the Accreditation Council for Continuing Medical Education (ACCME), indicates the following alert to medical professionals in its training curriculum:<br />
�Mortality is higher when the diagnosis of fungal infection is not made early. In one study, patients with lymphoma were housed in a protective environment &#8212; single-patient rooms under positive pressure with high-efficiency particulate air filtration&#8211; while undergoing bone marrow or peripheral stem cell transplantation.<br />
Despite these protective measures, 5.2% of patients developed nosocomial [hospitalization-related] aspergillosis. Although mortality rates among the patients with localized or pulmonary disease were substantial (42%), they were better than those of the patients who began treatment after the disease was already disseminated [spread to specific organs and/or tissues of the body). In fact, none of the patients with documented disseminated disease survived.�<br />
Immunosuppressed persons, persons with known autoimmune disease(s), and/or immune compromised persons who develop new or progressive pulmonary infiltrates while receiving broad-spectrum antibiotics, present major problems for differential diagnosis. These people are prime candidates for combined �opportunistic� fungal, bacterial, and viral infections and diseases with multiple medical complications. But, some evidence has surfaced recently that non-immune impaired individuals could also be at risk.<br />
Finally, if esteemed medical doctors at some of our country�s finest medical centers are telling their colleagues that certain environmental mold related illnesses are extremely hard to diagnose completely before treating, why do people in professional fields such as government, industrial hygiene, insurance, litigation, and others, who are not scientists by any stretch of the imagine in most cases still continuing to believe that this �mold� problem will fade with the wind.<br />
�Let me be perfectly clear,� to quote former president Richard M. Nixon, we are in the daring new frontier of molecular science! This is the 21st century, where advanced technology and the international Internet are bringing research and the world community closer together than ever before. This is a new era where �old politics� can no longer hide for long under the dark veil of secrecy! In this new advanced 21st century world science and medicine, two completely separate professions must accept greater responsibility in our safety and welfare. We are in a world at constant war against microbes that are all around us, that we live with, and live within us, and each and every human in order to live longer and healthier must also meet this educational demand by taking heed as to how to prevent these microbial beasts from silently stalking us as their prey, while we are still living.<br />
Exposed? Having health symptoms?  What tests or health concerns do we suggest you discuss with your medical doctor?<br />
If you or a loved one suspects that he or she has been exposed we suggest that you seek out a medical professional certified in Environmental or Occupational Health Medicine. Not always will you find a medical doctor that will agree with our suggested testing and medical evaluation information. We of course are not medical doctors, nor do we want to suggest in any way, that we propose to be authorities in the medical detection and treatment of mycotic diseases (mold related infections and other ailments). However, we will stand by the fact that the information we include in this next segment has helped many of the people whom have participated in the research behind this book obtain proper medical treatment for mold-related illnesses. We admit readily, that not all diseases are related to mold exposures and not all mold-related exposures are dangerous to animal or human health. The last thing anyone, especially we want to do, is cause fear to engulf anyone. Mold exposures to the CDC-38s mentioned herein can be dangerous to health, and can cause severe health problems in some susceptible individuals. Neither we nor the medical profession can accurately predict that you and your family members, if exposed to long-term, high-levels of exposures to dangerous �toxic molds,� have strong enough genetic and immune protection systems to keep you safe from mold �secondary chemicals� (mycotoxins) when disturbed. You are the first line of defense in protecting yourself and your living environment from mold contamination and infestation, but you do not have to be afraid of molds.<br />
If you have been exposed and are showing signs of chronic fatigue, having respiratory difficulties, and seem to get very sick when entering your home where mold has been discovered the following tests and physicians are �suggestive only� for initial health investigation as to the cause of your medical problems and/or health issues for subsequent discussion with your own medical doctor.<br />
Suggested Preliminary Medical Testing For High-Level Mold Exposures<br />
To better understand and aid in identifying or �ruling out� the potential for Systemic Fungal Disease (Mycotic Disease) as a diagnosis the following resources and blood serum testing processes are offered/suggested only. We are not physicians nor offer any position other in our capacity as Environmental Health Researchers:<br />
�	Primary Consultation: Merck Manual, 17th Edition, Chapter 158, Pg. 1209 (Other Chapters 76, 80, 113, 151, and157)<br />
� Professional Consultation/Referral: Dr. Vincent A. Marinkovich, M.D. (Diplomate, Am. Board of Allergy and Clinical Immunology), Graduate: CA Institute of Technology, Physics (1955), Harvard Medical School (1955), Pediatrics training John�s Hopkins Hospital (1959-61), Oxford University and King�s College London, Immunogenetics (1961-62), CA Institute of Technology, Specialized Immunology (1962-66), Brompton Research Hospital London, Fungal Hypersensitivity (1972-73), Founder and Chairman Allergy Testing Services (Owned currently by Hitachi Chemical Diagnostics, Inc. (1985-Present), Scientist/Clinical Associate Professor Stanford Medical School (1985-Present); 801 Brewster Avenue, Suite 220 Redwood City, CA 94063; Office: (650) 482-2800 or Fax: (650) 482-2802<br />
� Coordinate a blood serum test for Environmental MAST IgG Panel #16 with Hitachi Chemical Diagnostics, Inc., MAST Allergy Testing Service, 630 Clyde Court, Mountain View, CA 94043 Phone: (650) 961-5501 or 1-(800) 233-6278 and Fax (650) 969-2745. Test for 13 fungi species, dust mites, cockroach, etc.<br />
� Coordinate a blood serum test for Stachybotrys chartarum (black mold) with IBT Reference Laboratory 10453 West 84th Terrace, Lenexa, KS 66214. Phone 1-(800) 637-0370. Testing and ID Numbers are as follows: #1704 HSP Panel IgG, #1712 Stachybotrys Panel, and #2409 BRI Panel.<br />
� Coordinate Polymerase Chain Reaction (Genetic blood serum and saliva testing) with Immunosciences Lab, Inc., 8693 Wilshire Blvd., Beverly Hills, CA 90211. Phone: (310) 657-1077 or 1-(800) 950-4686, Fax: (310) 657-1053. They provide over 175 Molecular Medicine tests for detecting environmentally induced diseases relating to chemical-DNA adducts, specific gene mutation, and abnormal cell growth. You can also find their website on the Internet at: http://www.immuno-sci-lab.com for more information.</p>
<p>� Standard: CMV, EBV, IgG sub1-4 and normal RBC/WBC. For general guide in diagnosis consult Merck Manual, Coccidioidomycosis, Section: Diagnosis Pg. 1215<br />
Contaminated Homes And Health<br />
The myth that smaller and more confined spaces created by construction changes in building design are at fault for the sudden increase in construction defect cases involving water intrusion and mold infestation is just that, myth! Or at least generally stated as a myth. Mold problems involving private homes and public buildings have been an ongoing plague since man began living under covered shelter first recorded in biblical times. However in all fairness to this myth, newer construction with more confined spacing and building material changes designed to keep warm insulated air contained in the fall and winter months and cooler airflow inside in the spring and summer months has contributed to greater retention of condensation moisture, faster microbial infestation, increased levels and types of volatile organic compounds produced by microbes and other inorganic materials, and as such offers a greater potential for rapid deterioration of plumbing and household materials.<br />
In the mid-1980s, during double-digit inflation construction of new homes and commercial buildings called for less airflow per cubic feet per person, reducing the breathing space within a dwelling from about 20 cubic feet to approximately 5 cubic feet. It is also true that even though breathing space decreased the number of people sharing a smaller, more confined living or working quarters actually increased as building and maintenance costs escalated and the cost of living increased tremendously affecting in many venues of living and lifestyle. What is factual, is that private homes and commercial buildings constructed with building materials that molds use as nutrient sources when higher temperatures mix with higher indoor moisture certainly light a fire under the �Construction Defects� controversy leading to the present myth at hand. But, closer scrutiny into the background of molds and ecology reveals that many illnesses were caused by indoor mold contamination down throughout the ages, and this is problem of the ages and not strictly confined to one specific era in the evolution of mankind as we know it.<br />
In ecology, molds are perhaps the most dominate microbes to rule the earth. Nearly every species of life falls prey to molds, including bacteria and viruses. This is what the �Carbon Cycle,� or �Dust-to-Dust� in �layman�s� terms is all about. In the world of ecology it is the duty of molds to preserve their own species and to create life from the decay of life. Certain mold species are perhaps as old as the earliest stages of life formation and were genetically adapted to the dynamic physical and climatic rigors of environmental changes on earth long before animals, including man, ever stepped foot on living soil. In fact, nearly every living facet of man involves living, breathing microbes. Living cells are microbes in man that compose the immune system and every bodily organ and tissue belonging to man. It is therefore only natural that man would attract molds, bacteria, viruses and other microbial entities because animals and man have emerged as walking incubators for such inhabitation.</p>
<p>The peculiar bedfellows that man and microbes have become is intriguing at best, because herein we have species cohabitating in what scientists refer to as a �symbiotic� (or mutually beneficial and harmonious) state, but yet will ultimately end up in a bitter internal war in which molds always emerge as the victor. You see, molds do not die really; they just die until moisture is added to the right temperature and then they miraculously come back to life! Even if they are burned in a fire, they end up as minute particles that suspend in space until they are revived. Human ashes spread at sea reemerge as eventual mold and bacterial microbes to graze once again upon the earth.<br />
We realize that this seems like an awful lot of bad news to absorb! But transversely, the good news is that molds along with other microbes that are collectively known as �flora� actually orchestrate a harmonious life process that does not intentionally desire to wreak havoc on its animal and human hosts (as we are known in science). No&#8230;, what we as humans do is create certain external and internal environmental conditions out of lifestyle behavior and conditioning that weakens our body functioning to a point where molds begin to sense that their compatible environment is no longer safe and secure.</p>
<p>This might occur through consumption of microbial infested food, beverage, and organic products that serve as unhealthy nutrients which accumulatively signal the weakening and eventual death of body cells as our bodies start the aging process or gradually become ill. Additionally, changes to our external environment that allow mold species that normally do not adapt to or invade animal or human habitats might start to invade and take up temporary residence thereby disturbing the biological balance or as science states it, the �equilibrium� of nature that animal or man has adapted to and been regulated by throughout life.</p>
<p>So as we can surmise from all of this is that building construction modifications over the past two decades have contributed somewhat to the overall scope of increased incidents of illnesses referred to as �sick building syndrome� whereby the causes of various symptoms and ailments are not readily identified or known, or �building related illnesses� whereby the cause has been established and verified, but such modifications are not wholly responsible for the crisis that is emerging. So the question remains (to dispel the myth), what is causing us to suddenly realize the deadly health potential of molds that have been in and out of our bodies in large numbers for years. And, we do mean large numbers! It is estimated that we take in and release more than 16,000 quarts of air per day. Each quart averages approximately 70,000 visible and non-visible particles. In multiplying 16, 000 quarts times 70,000 particles in a 24 hour period, that sums to an astronomical figure of over 11, 200,000 billion particles consumed or absorbed daily.</p>
<p>What is even more remarkable is that the human body is in contact with over 3,000 pathogenic microbe species daily that potentially could kill a person swiftly, and yet the human immune system (which we have coined as the animal and human�s internal version of mold species) handles all of this with relative ease. That is, when biological harmony is synchronized and working according to plan. Another little tidbit of information is that the most harmful forms of microbes or microbial particles are three microns or less in size. The human eye cannot even see a microbe that size. In fact, the minimal visually adaptive size at which a microbe can be seen without the use of a microscope is at least 10 microns in diameter. To put all of this into prospective, the size of a general US postage stamp is 25,400 microns in size, and depending on the size of various microbes placed on it would be capable of holding from 1 million to a billion microbes within its borders. According to scientists studying such factors, approximately 98-99 percent of all microbial particles fall in the range of 5 microns in diameter or less. These particles tend to remain in suspension or settle so slowly that only high quality air cleaners are effective in removing them. Very small particles can remain in the air we breathe virtually until they are absorbed or inhaled by animals or humans.</p>
<p>So why is it that the legal system is overburdened with �mold litigation,� more so than ever before if it is not solely based on building products and materials and more confined construction?</p>
<p>It is because science over the last 16 years has literally advanced in coalition with advanced technology to literally a threshold equated to that of the �speed of light� in scientific terms. With the advent of highly advanced scientific instrumentation and testing devices scientists are now observing physically the genetic, metabolic and physical properties that less than two decades ago was unobtainable. Researchers are advancing and communicating around the globe �broad-based� through the use of the Internet and other communication entities on a moment-to-moment basis as opposed to annual conventions and symposiums of yesteryear. College and university students are being highly trained more in tune with this expansion and professors that once followed a strict outdated curriculum practice are now being forced to keep abreast of the latest scientific advancements in relating factually-based data academically, or face extinction in a world that is advancing with or without them. We are entering the 21st century of science of shared global information, and it is in and of itself bringing us closer to our own reality in dispelling the myths between microbes and our earthly environment.</p>
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